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Intra-tumour signalling entropy determines clinical outcome in breast and lung cancer

In agreement, meningococci deleted for IgA protease (Lin et al., 1997) or for HrpA (Tal et al., 2008) showed a dramatic reduction in survival/growth within different cell types

These findings suggest that CPT1C is a regulator of FA homeostasis in cancer cells and might be indirectly involved in the modulation of CPT1A activity through the lowering of cellular malonyl-CoA levels

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Rights and permissions About this article Cite this article Sinha, R., Singh, B