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Isothiocyanates react with certain regions on Keap1, eliminating Keap1s ability to target Nrf2 for degradation effectively serving the role of stressor

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showed that in cultured rat ventricular myocytes, trimetazidines beneficial effects were only observed when cells were undergoing hypoxia and not during normoxia, highlighting trimetazidine as a metabolic regulator that would not increase mitochondrial function in healthy states [123]