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The 50mg dose achieved the primary endpoint with pooled 16.4% placebo-adjusted clinical remission (p 1 at week 12, with secondary endpoints including clinical response, endoscopic remission, and safety measures

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Characterization of the phenotypes of GLP-1R neurons in areas directly targeted by semaglutide revealed that proteins known to be involved in appetite regulation coexpress with GLP-1R

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GLP-1 inhibits FoxO1, promoting -cell proliferation, and enhances PDX1 activity, thereby stimulating cellular proliferation