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IGF1-LR3, a modified version of insulin-like growth factor-1, has a longer half-life than standard IGF-1 because it does not bind to IGF-1 binding proteins well

This is a general risk pattern whenever a treatment is unapproved, lacks strong human safety data, and is sourced outside regulated channels

(1970) Mechanisms of IGF-1-Mediated Regulation of Skeletal Muscle Hypertrophy and Atrophy National Institute of Diabetes and Digestive and Kidney Diseases (2018) Tesamorelin Vauga P, Kuma M, Stojkoviov D, et al

Addressing deficiencies in these nutrients supports sermorelin's mechanism of action

Yang H, Kuhn C, Kolben T, et al