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A number of factors cause Th17 and Th2 cells to produce excessive inflammation including penetration of the intestinal epithelium by gut microbes, composition of the intestinal microbiota, injury to the intestinal wall, insufficient mucus layer production, and allergies or sensitivities to foods

The identified markers should therefore be considered hypothesis-generating and warrant further investigation in larger, prospective studies with longitudinal follow-up

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These compounds could be possible agents for treating hyperuricemia and gout [75]