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Exenatide treatment ameliorates these morphological alterations and inhibits the mitochondrial apoptotic pathway by suppressing cytochrome c release and favorably modulating the expression of pro-apoptotic (Bax, caspase-3) and anti-apoptotic (Bcl-2) proteins, thereby protecting RGC-5 cells from hyperglycemic injury [43]

In wt cells, the OxD values differed only slightly between the three compartments and deletion of GLR1 resulted in increased OxD values at steady state in all three compartments (Figure 3B)

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The active ingredient semaglutide was approved by the U.S