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10.1002/pbc.25994 [DOI] [PMC free article] [PubMed] [Google Scholar] Wang X.J., Hayes J.D., Henderson C.J., and Wolf C.R

Basing on the secondary injury mechanism, anti-oxidation and anti-excitatory amino have been used in releasing the neurological dysfunction, not showing effect, and currently, there are no effective treatments approved by clinical trials for TBI patients (Pearn et al., 2017)

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Neurodevelopmental low-dose bisphenol A exposure leads to early life-stage hyperactivity and learning deficits in adult zebrafish