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[1] This review focuses on glutathione as a skin-lightening agent

Moreover, short- and long-term tunicamycin treatments that trigger ER stress via UDP-HexNAc inhibition were ineffective against ferroptosis (Supplementary Fig

Intrathyroidal T lymphocytes, macrophages, and Th1 cytokines contribute to elevated Fas expression and procaspase levels

Clinical alterations resulting from these mutations are hypothesized to be derived from either loss or gain of function of GCase and interaction with its substrates like glycosphingolipids (GSLs), glucosylceramide (GlcCer) and glucosylsphingosine (GlcSph)

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