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Current understanding of PD pathogenesis implicates neurofilaments, synaptic vesicle proteins, and ubiquitinated -synuclein as primary contributors to the disease pathology (161)

In vitro , the viability of human astrocytes is improved with 15 mmol/L glucose treatment, but astrocytes are damaged by 30 mmol/L glucose, with changes in the morphology and expression of cytoskeletal proteins, such as GFAP and vimentin

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The most clinically relevant efficacy data came from METAOD005, a 12-week randomized double-blind placebo-controlled study