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Close observation helps manage adverse reactions swiftly and ensures the treatment remains effective

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Further studies revealed that DAA was able to bind to Keap1 in the cytoplasm and increase the luciferase activity of Nrf2-antioxidant reactive element (ARE), promoting the expression of downstream antioxidant gene hame oxygenase-1 (HO-1), GSH, and GPx4 through the Keap1/Nrf2 pathway, while DAA treatment results in achieving attenuation of ROS and lipid peroxidation ( 2+ , which is manifested as an inhibition of ferroptosis (66)

Higher concentrations are typically reserved for patients who have plateaued on 5% or who have extensive thinning

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