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These results suggest that the dimer form could be a more effective GSTP1 inhibitor, possibly due to stronger binding interactions within the GSTP1 active site

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302 Myeloid cells can activate their own Arg 1 expression in response to tumor-derived GM-CSF stimulation through activation of STAT3, p38, and cAMP signaling pathways, and Arg 1-expressing myeloid cells depleting environmental arginine inhibits T cell function

coli cells per liter of Luria-Bertani (LB) broth media, with around 50 g of E

doi: 10.1056/nejm200101043440106 72 DworskiR