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In support to this notion, we have shown: (i) L-cysteine, an activator of H 2 S producing enzymes (CSE and CBS), or the exogenous H 2 S donors NaHS and GYY4137 inhibited OA-induced GLP-1 and PYY release, and (ii) inhibition of release is due to suppression of the Epac/PLC-/Ca 2+ pathway downstream of cAMP pathway

To view a copy of this licence, visit About this article Cite this article Belachew, E.A., Peterson, G.M

Evaluation of the diagnostic potential of urinary n-Acetyltyramine-O,beta-glucuronide (NATOG) as diagnostic biomarker for Onchocerca volvulus infection

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More in-depth research that clarifies the roles of these components of the GST-detoxification system are much needed, in order to produce compounds that have minimal side effects and high GST selectivity