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Deisseroth, A, Kantarjian H, Andreeff M , Talpaz M, Keating M, Khouri I, Champlin R
The inflammatory microenvironment contributes to prostate cancer initiation, promotion, and progression via a number of mechanisms, including increased levels of specific cytokines/chemokines [38]
As the GLP-1R is not expressed on human hepatocytes, the GLP-1RA-mediated effects on fatty liver are probably indirectly mediated through GLP-1RA-induced weight loss and ensuing improvement in insulin resistance, and, potentially, anti-inflammatory effects associated with GLP-1RA treatment (29)
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