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NO-dependent oxidative stress and endothelial dysfunction are crucial factors contributing to cardiovascular pathophysiology in the PCOS setting, where generated reactive oxygen species cause uncoupling of endothelial nitric oxide synthase (eNOS), decreasing NO synthesis, which ultimately induces endothelial dysfunction and cardiac injury [41]

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Yet the govenment keeps on increasing funding for DDAs with no concern for transparency and accountability

Betaine is converted to TMA through the action of betaine reductase