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Study 7: The only published clinical data in humans Citation: Veljaca M., Pavic Sladoljev D., Mildner B., et al

Our major insight from examining the vitamin B 12 GSH data in conjunction with the Reed-Nijhout model is that cysteine-block explains why GSH does not increase as a result of vitamin B 12 deficiency induced hyperhomocysteinemia

The result could not be fully confirmed as there are some limitations

Several lines of evidence point to upregulation of vascular endothelial growth factor receptor 2 (VEGFR2) signaling and modulation of the nitric oxide system [Huang et al., Vascular Pharmacology, 2015]

In many cases, our discomfort and suffering come down to estrogen dominance and MTHFR