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While GLP-1R is known to be expressed in the central nervous system [55], the effectiveness of GLP-1 analogs in patients with MC4R-POMC pathway deficiency has not been well established

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Basing on the secondary injury mechanism, anti-oxidation and anti-excitatory amino have been used in releasing the neurological dysfunction, not showing effect, and currently, there are no effective treatments approved by clinical trials for TBI patients (Pearn et al., 2017)

Oxidative stress is caused due to an imbalance between production of reactive oxygen species (free radicals) and effectiveness of antioxidant defense

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