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Furthermore, UA functions in the central nervous system (CNS) as an antioxidant, guarding against acute stroke and Parkinsons disease [126]

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Agm antidepressant effects have been reported to depend upon both imidazoline and GABA receptors, and NMDA-NO axis inhibition (and less consistently between studies, perhaps also serotonin and adrenergic receptors), suggesting a complex ketamine-like rapid antidepressant mechanism that may involve Agm/Glu co-transmission (Uzbay, 2012

doi: 10.1021/acs.jproteome.1c00851

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