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These beneficial effects include the counteractions of traumatic brain injury and severe encephalopathies after NSAID overdose, insulin overdose, magnesium overdose, and exposure to the neurotoxin cuprizone in a rat model of multiple sclerosis [33,34,35,36,37,38,39,40,41]

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NRF1 is typically responsible for activating the expression of TFAM, thereby increasing its concentration in the mitochondria (Fig

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SIRT1 exhibits broad multi-pathway regulatory capacity, making interventions targeting SIRT1 a key focus in current MASLD research