doi: 10.1021/acs.jafc.8b01074 41
Quartarone A, Bagnato S, Rizzo V, Siebner HR, Dattola V, Scalfari A, et al
69 In gastric cancer cells, the absence of CDO1 contributed to restore cellular glutathione levels, to prevent ROS and lipid peroxidation, and to promote resistance to ferroptosis, a type of programmed cell death dependent on iron and associated with lipid peroxide accumulation
That framing is more useful than asking which peptide is best, because the better choice depends on the type of repair-focused research you are actually interested in
(Midi does not prescribe this or other unapproved peptides.) What the science says: Theres promising preclinical (aka animal or lab) data on MOTS-c, and early research suggests that MOTS-c may improve metabolism by activating the AMPK pathway and help reverse age-related insulin resistance