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For any patients presenting with compatible illness, inform the Victorian Poisons Information Centre on 13 11 26 who can provide clinical and specific testing advice

All presented information is based on scientific publications which can be found at the end of product description below

This mechanism involves: Sequestration of monomeric actin (G-actin) to prevent premature polymerization Promotion of actin polymerization into functional microfilaments (F-actin) when needed Enhanced cellular migration through cytoskeletal reorganization Facilitation of cell shape changes necessary for wound healing Research indicates TB-500 may work with the Arp2/3 protein complex to orchestrate actin polymerization, though this mechanism requires further clarification[4]