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Niesman IR, Zemke N, Fridolfsson HN, et al

Research in preclinical models suggests that AOD 9604 may support lipolysis by upregulating beta-3 adrenergic receptor expression and inhibiting new fat cell formation, with human trial data confirming no IGF-1 elevation or glucose disruption [2, 3]

Role of microbiota short-chain fatty acids in the pathogenesis of autoimmune diseases